Cellular Regulation of the Inflammatory Response

The acute inflammatory response and its regulation.

The acute inflammatory response is composed of an elaborate cascade of both proinflammatory and anti-inflammatory mediators. The balance between these mediators often determines the outcome after injury. In clinical scenarios, such as trauma or sepsis, there is often unregulated production of proinflammatory mediators that can cause multiple organ failure. Further understanding of the endogenou...

a tripartite model of efl teachers attributions, burnout, and ‎self-regulation: towards the prospects of effective teaching

همطالعه حاضر به ارائه مدلی برای آموزش موثر زبان انگلیسی ‏می پردازد. مدل حاضر از سه عامل تاثیر گذار در کارایی ‏تدریس معلمان زبان انگلیسی بهره می برد. این سه عامل شامل ‏سبکهای اسنادی، خود تنطیمی و فرسودگی شغلی معلمان ایرانی ‏زبان انگلیسی می باشد. رساله مورد نظر درچهار فاز طراحی ‏شده است: فاز اول شامل طراحی و رواسازی پرسشنامه سبکهای ‏اسنادی معلمان زبان انگلیسی و فاز دوم شامل استفاده از ‏این پرسشنا...

The cellular inflammatory response in human spinal cords after injury.

Spinal cord injury (SCI) provokes an inflammatory response that generates substantial secondary damage within the cord but also may contribute to its repair. Anti-inflammatory treatment of human SCI and its timing must be based on knowledge of the types of cells participating in the inflammatory response, the time after injury when they appear and then decrease in number, and the nature of thei...

NGF and Its Receptors in the Regulation of Inflammatory Response

There is growing interest in the complex relationship between the nervous and immune systems and how its alteration can affect homeostasis and result in the development of inflammatory diseases. A key mediator in cross-talk between the two systems is nerve growth factor (NGF), which can influence both neuronal cell function and immune cell activity. The up-regulation of NGF described in inflame...

Chronic Inflammatory Conditions Enhancing Neutrophil Infiltration under Regulation of the Inflammatory Response: